Influence of pregnancy on human immunodeficiency virus disease
Identifieur interne : 001519 ( Main/Exploration ); précédent : 001518; suivant : 001520Influence of pregnancy on human immunodeficiency virus disease
Auteurs : Alain Berrebi [France] ; Wilhelm E. Kobuch [France] ; Jacqueline Puel [France] ; Joelle Tricoire [France] ; Philippe Herne [France] ; Helene Grandjean [France] ; George Pontonnier [France]Source :
- European Journal of Obstetrics & Gynecology and Reproductive Biology [ 0301-2115 ] ; 1990.
Descripteurs français
- Wicri :
- topic : Conférence internationale.
English descriptors
- KwdEn :
- Abstract volume, Clinical stage, Clinical stages, Control group, Control populations, Disease progression, First year, Generalized lymphadenopathy, Gynecol, Human immunodeficiency virus, Human pregnancy, Immunodeficiency, Immunodeficiency syndrome, International conference, Laboratory data, Laboratory parameters, Leucocyte count, Lymphocyte, Obstet gynecol, Poster presentation, Pregnancy, Pregnant patients, Pregnant women, Risk factor, Second trimester, Second year, Sedimentation rate, Statistical analysis, Study period, Syndrome, Third year, Time point.
- Teeft :
- Abstract volume, Clinical stage, Clinical stages, Control group, Control populations, Disease progression, First year, Generalized lymphadenopathy, Gynecol, Human immunodeficiency virus, Human pregnancy, Immunodeficiency, Immunodeficiency syndrome, International conference, Laboratory data, Laboratory parameters, Leucocyte count, Lymphocyte, Obstet gynecol, Poster presentation, Pregnancy, Pregnant patients, Pregnant women, Risk factor, Second trimester, Second year, Sedimentation rate, Statistical analysis, Study period, Syndrome, Third year, Time point.
Abstract
Over a period of 3 years (mean 16, extremes 3 and 36 months), we compared clinical and laboratory parameters of 128 female, human immunodeficiency virus (HIV)-infected patients, all in clinical stage II or III (CDC classification). 34 patients were pregnant and delivered a viable infant after at least 28 weeks of amenorrhea (group I), 29 patients were pregnant and had a spontaneous or induced abortion during the first or second trimester (group II), and 64 were non-pregnant female control patients (group III). The changes in the clinical stages over time were not statistically significant between the groups. The only laboratory parameters that were significantly higher in group I at the time of the delivery were: leucocyte count (p < 0.001), lymphocyte count (p < 0.05), and sedimentation rate (p < 0.001). These changes are known to be related to pregnancy and not to HIV disease. All other laboratory parameters showed no significant differences within and between the groups. We conclude, that pregnancy — carried to term or interrupted — does not aggravate the natural evolution of HIV infection in clinical stage II and III patients.
Url:
DOI: 10.1016/0028-2243(90)90027-X
Affiliations:
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Le document en format XML
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<front><div type="abstract" xml:lang="en">Over a period of 3 years (mean 16, extremes 3 and 36 months), we compared clinical and laboratory parameters of 128 female, human immunodeficiency virus (HIV)-infected patients, all in clinical stage II or III (CDC classification). 34 patients were pregnant and delivered a viable infant after at least 28 weeks of amenorrhea (group I), 29 patients were pregnant and had a spontaneous or induced abortion during the first or second trimester (group II), and 64 were non-pregnant female control patients (group III). The changes in the clinical stages over time were not statistically significant between the groups. The only laboratory parameters that were significantly higher in group I at the time of the delivery were: leucocyte count (p < 0.001), lymphocyte count (p < 0.05), and sedimentation rate (p < 0.001). These changes are known to be related to pregnancy and not to HIV disease. All other laboratory parameters showed no significant differences within and between the groups. We conclude, that pregnancy — carried to term or interrupted — does not aggravate the natural evolution of HIV infection in clinical stage II and III patients.</div>
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